Prof. Dr. BAHAR TUNÇTAN
- Yöksis ID 3939
- Researcher ID G-3428-2015
- ORCID ID 0000-0003-3439-7803
- Scopus ID
- Scholar ID mLP60wUAAAAJ&hl
- E-posta btunctan@mersin.edu.tr
- Dahili Telefon +90 324 361 00 01 - 12121
Birim
ECZACILIK FAKÜLTESİ
ECZACILIK MESLEK BİLİMLERİ BÖLÜMÜ
FARMAKOLOJİ ANABİLİM DALI
ECZACILIK MESLEK BİLİMLERİ BÖLÜMÜ
FARMAKOLOJİ ANABİLİM DALI
ÜAK Temel Alan Bilgisi
Sağlık Bilimleri Temel Alanı
Farmakoloji
Farmakoloji
Son 5 Makale
RIPK1/RIPK3/MLKL Necrosome Contributes to the Sepsis-induced Cardiorenal Necroptotic Inflammatory Injury and Mortality
CURRENT MOLECULAR PHARMACOLOGY, vol. 17, no. , pp. – Link 2025
Urotensin-II receptor contributes to the pro-inflammatory TLR4/MyD88/NF-κB/iNOS/NO pathway-mediated cardiovascular response to systemic lipopolysaccharide challenge in a septic shock model in rats
Cellular and Molecular Biology, vol. 71, no. 5, pp. – Link 2025
RIPK1/RIPK3/MLKL necrosome contributes to the sepsis-induced cardiorenal necroptotic inflammatory injury and mortality
Current Molecular Pharmacology, vol. 17, no. , pp. – Link 2024
Inhibition of RIPK1-driven necroptosis ameliorates inflammatory hyperalgesia caused by lipopolysaccharide: involvement of TLR-, NLRP3-, and caspase-11- mediated signaling pathways
Cellular and Molecular Biology, vol. 70, no. 12, pp. 52–58 Link 2024
Protection by selective mTORC2 inhibition of Zymosan-induced hypotension and systemic inflammation mediated via IKKα/IκB-α/ NF-κB activation
PROSTAGLANDINS & OTHER LIPID MEDIATORS, vol. 175, no. , pp. 106918– Link 2024
CURRENT MOLECULAR PHARMACOLOGY, vol. 17, no. , pp. – Link 2025
Urotensin-II receptor contributes to the pro-inflammatory TLR4/MyD88/NF-κB/iNOS/NO pathway-mediated cardiovascular response to systemic lipopolysaccharide challenge in a septic shock model in rats
Cellular and Molecular Biology, vol. 71, no. 5, pp. – Link 2025
RIPK1/RIPK3/MLKL necrosome contributes to the sepsis-induced cardiorenal necroptotic inflammatory injury and mortality
Current Molecular Pharmacology, vol. 17, no. , pp. – Link 2024
Inhibition of RIPK1-driven necroptosis ameliorates inflammatory hyperalgesia caused by lipopolysaccharide: involvement of TLR-, NLRP3-, and caspase-11- mediated signaling pathways
Cellular and Molecular Biology, vol. 70, no. 12, pp. 52–58 Link 2024
Protection by selective mTORC2 inhibition of Zymosan-induced hypotension and systemic inflammation mediated via IKKα/IκB-α/ NF-κB activation
PROSTAGLANDINS & OTHER LIPID MEDIATORS, vol. 175, no. , pp. 106918– Link 2024
Son 5 Bildiri
RIPK1/RIPK3/MLKL necrosome contributes to the nitrosative stress-mediated cardiorenal injury and mortality in lipopolysaccharide-induced sepsis model in mice (Lipopolisakkarit ile farelerde oluşturulan sepsis modelinde nitrozatif stres aracılıklı enflamatuvar kardiyorenal zedelenme ve mortaliteye RIPK1/RIPK3/MLKL nekrozomu katkıda bulunmaktadır)
16th International Medical and Health Sciences Research Congress (16. Uluslararası Tıp ve Sağlık Bilimleri Araştırmaları Kongresi) (06.07.2024 - 07.07.2024) Link 2024
Urotensin-II receptor contributes to the nitric oxide-mediated hypotension in lipopolysaccharide-induced septic shock model in rats (Lipopolisakkarit ile sıçanlarda oluşturulan septik şok modelinde nitrik oksit aracılıklı hipotansiyona ürotensin-II reseptörü katkıda bulunmaktadır)
16th International Medical and Health Sciences Research Congress (16. Uluslararası Tıp ve Sağlık Bilimleri Araştırmaları Kongresi) (06.07.2024 - 07.07.2024) Link 2024
Farelerde lipopolisakkaritin neden olduğu enflamatuvar hiperaljeziyi RIP1 inhibitörü nekrostatin-1s önlemektedir (RIPK1 inhibitor necrostatin-1s prevents lipopolysacharide-induced inflammatory hyperalgesia in mice)
27. Ulusal ve 2. Uluslararası Farmakoloji Kongresi (27th National and 2st International Pharmacology Congress) (23.11.2023 - 26.11.2023) Link 2024
Sıçanlarda lipopolisakkarit ile oluşturulan septik şok modelinde iNOS aracılıklı hipotansiyona vasküler RIPK1/RIP3/MLKL nekrozomunun etkinleşmesi katkıda bulunmaktadır (Activation of vascular RIPK1/RIPK3/MLKL necrosome contributes to the iNOS-mediated hypotension in lipopolysaccharide-induced septic shock model in rats)
27. Ulusal ve 2. Uluslararası Farmakoloji Kongresi (27th National and 2st International Pharmacology Congress) (23.11.2023 - 26.11.2023) Link 2024
Protective effects of selective mTORC2 inhibition on zymosan-induced hypotension in a rat model of non-septic shock: Contribution of IKKα/IκB-α/NF-κB pathway.
9th International BAU Drug Design Congress (29.12.2023 - 29.12.2023) Link 2023
16th International Medical and Health Sciences Research Congress (16. Uluslararası Tıp ve Sağlık Bilimleri Araştırmaları Kongresi) (06.07.2024 - 07.07.2024) Link 2024
Urotensin-II receptor contributes to the nitric oxide-mediated hypotension in lipopolysaccharide-induced septic shock model in rats (Lipopolisakkarit ile sıçanlarda oluşturulan septik şok modelinde nitrik oksit aracılıklı hipotansiyona ürotensin-II reseptörü katkıda bulunmaktadır)
16th International Medical and Health Sciences Research Congress (16. Uluslararası Tıp ve Sağlık Bilimleri Araştırmaları Kongresi) (06.07.2024 - 07.07.2024) Link 2024
Farelerde lipopolisakkaritin neden olduğu enflamatuvar hiperaljeziyi RIP1 inhibitörü nekrostatin-1s önlemektedir (RIPK1 inhibitor necrostatin-1s prevents lipopolysacharide-induced inflammatory hyperalgesia in mice)
27. Ulusal ve 2. Uluslararası Farmakoloji Kongresi (27th National and 2st International Pharmacology Congress) (23.11.2023 - 26.11.2023) Link 2024
Sıçanlarda lipopolisakkarit ile oluşturulan septik şok modelinde iNOS aracılıklı hipotansiyona vasküler RIPK1/RIP3/MLKL nekrozomunun etkinleşmesi katkıda bulunmaktadır (Activation of vascular RIPK1/RIPK3/MLKL necrosome contributes to the iNOS-mediated hypotension in lipopolysaccharide-induced septic shock model in rats)
27. Ulusal ve 2. Uluslararası Farmakoloji Kongresi (27th National and 2st International Pharmacology Congress) (23.11.2023 - 26.11.2023) Link 2024
Protective effects of selective mTORC2 inhibition on zymosan-induced hypotension in a rat model of non-septic shock: Contribution of IKKα/IκB-α/NF-κB pathway.
9th International BAU Drug Design Congress (29.12.2023 - 29.12.2023) Link 2023